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Author Duan, Y.; Wang, Z.; Zhang, H.; He, Y.; Fan, R.; Cheng, Y.; Sun, G.; Sun, X. url  doi
openurl 
  Title Extremely low frequency electromagnetic field exposure causes cognitive impairment associated with alteration of the glutamate level, MAPK pathway activation and decreased CREB phosphorylation in mice hippocampus: reversal by procyanidins extracted from the lotus seedpod Type Journal Article
  Year 2014 Publication Food & Function Abbreviated Journal  
  Volume 5 Issue Pages 2289-2289  
  Keywords  
  Abstract  
  Address  
  Corporate Author Thesis  
  Publisher Royal Society of Chemistry Place of Publication Editor  
  Language Summary Language Original Title  
  Series Editor Series Title Abbreviated Series Title  
  Series Volume Series Issue Edition  
  ISSN ISBN Medium  
  Area WP5 In vitro Expedition Conference  
  Notes Approved no  
  Call Number UNIBAS @ david.schuermann @ Serial 616  
Permanent link to this record
 

 
Author Winklhofer, M. url  doi
openurl 
  Title An Avian Magnetometer Type Journal Article
  Year 2012 Publication Science Abbreviated Journal Science  
  Volume 336 Issue 6084 Pages 991-992  
  Keywords  
  Abstract Homing pigeons have remarkable navigational
skills that allow them to
fi nd their way back to the loft when
released from an unfamiliar location hundreds
of miles away. To perform such a feat,
they rely on various cues, such as odors and
Earth’s magnetic fi eld ( 1, 2). Yet, how birds
and other animals obtain magnetic-fi eld information
has been a mystery. On page 1054 of
this issue, Wu and Dickman ( 3) report how
this information is neurally encoded and suggest
a candidate magnetic sensory organ in
the inner ear of the pigeon ( 4).
 
  Address  
  Corporate Author Thesis  
  Publisher Place of Publication Editor  
  Language Summary Language Original Title  
  Series Editor Series Title Abbreviated Series Title  
  Series Volume Series Issue Edition  
  ISSN 0036-8075 ISBN Medium  
  Area WP6 In vivo Expedition Conference  
  Notes Approved no  
  Call Number CBM.UAM @ ccobaleda @ Serial 188  
Permanent link to this record
 

 
Author Hirota, T.; Lee, J.W.; St. John, P.C.; Sawa, M.; Iwaisako, K.; Noguchi, T.; Pongsawakul, P.Y.; Sonntag, T.; Welsh, D.K.; Brenner, D.A.; Doyle, F.J.; Schultz, P.G.; Kay, S.A. url  doi
openurl 
  Title Identification of Small Molecule Activators of Cryptochrome Type Journal Article
  Year 2012 Publication Science Abbreviated Journal Science  
  Volume 337 Issue 6098 Pages 1094-1097  
  Keywords  
  Abstract  
  Address  
  Corporate Author Thesis  
  Publisher Place of Publication Editor  
  Language Summary Language Original Title  
  Series Editor Series Title Abbreviated Series Title  
  Series Volume Series Issue Edition  
  ISSN 0036-8075 ISBN Medium  
  Area Expedition Conference  
  Notes Approved no  
  Call Number IT'IS @ evaj @ Serial 449  
Permanent link to this record
 

 
Author Lu, Y.; He, M.; Zhang, Y.; Xu, S.; Zhang, L.; He, Y.; Chen, C.; Liu, C.; Pi, H.; Yu, Z.; Zhou, Z. url  openurl
  Title Differential Pro-Inflammatory Responses of Astrocytes and Microglia Involve STAT3 Activation in Response to 1800 MHz Radiofrequency Fields Type Journal Article
  Year 2014 Publication PloS one Abbreviated Journal  
  Volume 9 Issue 9 Pages e108318-e108318  
  Keywords  
  Abstract Microglia and astrocytes play important role in maintaining the homeostasis of central nervous system (CNS). Several CNS impacts have been postulated to be associated with radiofrequency (RF) electromagnetic fields exposure. Given the important role of inflammation in neural physiopathologic processes, we investigated the pro-inflammatory responses of microglia and astrocytes and the involved mechanism in response to RF fields. Microglial N9 and astroglial C8-D1A cells were exposed to 1800 MHz RF for different time with or without pretreatment with STAT3 inhibitor. Microglia and astrocytes were activated by RF exposure indicated by up-regulated CD11b and glial fibrillary acidic protein (GFAP). However, RF exposure induced differential pro-inflammatory responses in astrocytes and microglia, characterized by different expression and release profiles of IL-1β, TNF-α, IL-6, PGE2, nitric oxide (NO), inducible nitric oxide synthase (iNOS) and cyclooxygenase 2 (COX2). Moreover, the RF exposure activated STAT3 in microglia but not in astrocytes. Furthermore, the STAT3 inhibitor Stattic ameliorated the RF-induced release of pro-inflammatory cytokines in microglia but not in astrocytes. Our results demonstrated that RF exposure differentially induced pro-inflammatory responses in microglia and astrocytes, which involved differential activation of STAT3 in microglia and astrocytes. Our data provide novel insights into the potential mechanisms of the reported CNS impacts associated with mobile phone use and present STAT3 as a promising target to protect humans against increasing RF exposure.  
  Address  
  Corporate Author Thesis  
  Publisher Place of Publication Editor  
  Language Summary Language Original Title  
  Series Editor Series Title Abbreviated Series Title  
  Series Volume Series Issue Edition  
  ISSN ISBN Medium  
  Area WP5 In vitro Expedition Conference  
  Notes Approved no  
  Call Number UNIBAS @ david.schuermann @ Serial 555  
Permanent link to this record
 

 
Author Reale, M.; Kamal, M. a; Patruno, A.; Costantini, E.; D'Angelo, C.; Pesce, M.; Greig, N.H. url  doi
openurl 
  Title Neuronal cellular responses to extremely low frequency electromagnetic field exposure: implications regarding oxidative stress and neurodegeneration Type Journal Article
  Year 2014 Publication PloS one Abbreviated Journal  
  Volume 9 Issue 8 Pages e104973-e104973  
  Keywords  
  Abstract Neurodegenerative diseases comprise both hereditary and sporadic conditions characterized by an identifying progressive nervous system dysfunction and distinctive neuopathophysiology. The majority are of non-familial etiology and hence environmental factors and lifestyle play key roles in their pathogenesis. The extensive use of and ever increasing worldwide demand for electricity has stimulated societal and scientific interest on the environmental exposure to low frequency electromagnetic fields (EMFs) on human health. Epidemiological studies suggest a positive association between 50/60-Hz power transmission fields and leukemia or lymphoma development. Consequent to the association between EMFs and induction of oxidative stress, concerns relating to development of neurodegenerative diseases, such as Alzheimer disease (AD), have been voiced as the brain consumes the greatest fraction of oxygen and is particularly vulnerable to oxidative stress. Exposure to extremely low frequency (ELF)-EMFs are reported to alter animal behavior and modulate biological variables, including gene expression, regulation of cell survival, promotion of cellular differentiation, and changes in cerebral blood flow in aged AD transgenic mice. Alterations in inflammatory responses have also been reported, but how these actions impact human health remains unknown. We hence evaluated the effects of an electromagnetic wave (magnetic field intensity 1mT; frequency, 50-Hz) on a well-characterized immortalized neuronal cell model, human SH-SY5Y cells. ELF-EMF exposure elevated the expession of NOS and O2-, which were countered by compensatory changes in antioxidant catylase (CAT) activity and enzymatic kinetic parameters related to CYP-450 and CAT activity. Actions of ELF-EMFs on cytokine gene expression were additionally evaluated and found rapidly modified. Confronted with co-exposure to H2O2-induced oxidative stress, ELF-EMF proved not as well counteracted and resulted in a decline in CAT activity and a rise in O2- levels. Together these studies support the further evaluation of ELF-EMF exposure in cellular and in vivo preclinical models to define mechanisms potentially impacted in humans.  
  Address  
  Corporate Author Thesis  
  Publisher Place of Publication Editor  
  Language Summary Language Original Title  
  Series Editor Series Title Abbreviated Series Title  
  Series Volume Series Issue Edition  
  ISSN ISBN Medium  
  Area WP5 In vitro Expedition Conference  
  Notes Approved no  
  Call Number UNIBAS @ david.schuermann @ Serial 625  
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